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Understanding Bursitis and Bursitis Treatments

Understanding Bursitis and Bursitis Treatments

by Dr. Rick Boatright, author of "Surviving Type II Diabetes"
Bursitis is a medical term derived from two words: Bursa – which is a small, fluid-filled sack over a joint that helps it to move smoothly with reduced friction; and “itis” which is a medical suffix meaning inflamed or sore. So the first and most important thing to understand about bursitis is that it is certainly not any kind of a disease process that attacks a joint. Bursitis is simply a medical term stating that the bursa is sore, period.
Once a doctor had moved your joint around, examined it and determined that the “diagnosis” is bursitis, the question becomes - What can be done about it.
A medical doctor is an allopathic doctor. The definition of an allopathic doctor is one who sees his or her job as interfering with the symptoms in order to make the patient feel better. Of course, we all want to feel better! But an allopathic approach to treating bursitis is usually a matter of prescribing anti-inflammatory, and/or pain medications. The patient will almost certainly feel better, at least temporarily; however, such treatment addresses only effects and not the underlying cause.
A bursa doesn’t get sore without any reason at all. It gets sore because something is physically irritating it! Treating the symptom is fine for temporary relief, but what’s even more important is removing the source of the irritation!
One could compare it to a scenario where a patient goes to a doctor with an arrow in his back. The doctor diagnoses a puncture wound and prescribes pain killers, antibiotics and an anti-inflammatory and then sends the patient home, but with the arrow still in his back.
Joints that are covered by bursas, such as shoulders, knees and hips, are vulnerable to subtle misalignments that are often difficult to see on X-rays, but fairly easy for many chiropractors to identify. These misalignments cause the joint to be stressed unevenly and to introduce continuous irritation into the associated bursa. Quite often, when the joint motion is corrected and the irritation thus removed from the bursa, the bursitis – sore bursa – resolves on its own without medications or painful physical therapy.
So don’t be bamboozled into believing that some disease called bursitis has attacked your joints. The concept is ludicrous! Bursitis simply means a sore bursa. Identify what’s causing it to be sore and correct it. Don’t just treat with comfort drugs while leaving the arrow in the back!

What Is Arthritis?

What is Arthritis?

by Dr. Rick Boatright, author of "Surviving Type II Diabetes"
The word arthritis is composed of two root words – arthro, which means joint and itis, which means sore or irritated. So it’s important to understand that arthritis is not a disease. It never has been and never will be. Arthritis is simply a description of a sore joint. You tell the doctor, “My knee’s sore.” He says, “You have arthritis.” You’ve said exactly the same thing. You said it in English and he said it in Latin. You already know that the joint is sore. You want to know why.
The ‘why’ part is indicated by the word preceding the word arthritis. So when you see ‘rheumatoid arthritis,’ you have a sore joint (arthritis) being caused by a rheumatoid condition in which the body’s immune system recognizes its own joint fluids as foreign invaders and the body is trying to defend against them. In ‘psoriatic arthritis’ there’s pain in the joint (arthritis) caused by psoriasis inside the joint itself.
Osteoarthritis is somewhat unique, even though it’s the most common form of ‘arthritis.’ There is joint pain (arthritis) being caused by … ‘osteo?’ Osteo simply means bone. So osteoarthritis is joint pain caused by no disease process at all, but by “bone.” Investigation shows that THE cause of osteoarthritis is excess wear of a joint. The excess wear of the joint causes pain (arthritis) and eventual destruction of the joint.
But what’s really interesting is that runners don’t get more arthritis in their knees than couch potatoes, so it’s certainly not a matter of how many times we move a joint in our lives or how much we pound them with activities like running. And when people get osteoarthritis, one of its distinguishing characteristics is that it’s unilateral, meaning it will be in one knee but not the other, or in one hip but not the other or in one vertebra but all of them. This is another indicator that it’s not a disease because a disease process would be the same joint on both sides.
If neither the number of times we use a joint nor how hard we use it isn’t the cause of excess wear, what IS the cause?
According to studies, a joint wears up to seven times faster when it’s misaligned! This accounts for why one joint can have severe osteoarthritis, even to the point of needing joint replacement, but the other functions just fine.
Chiropractors have been experts at proper joint alignment since 1895! If you want to stop the progression of osteoarthritis, try chiropractic. If it hasn’t progressed past a certain degree of destruction, it can even be reversed and in most cases the pain can be minimized or even resolved.

What Causes Type 2 Diabetes?

What Causes Type 2 Diabetes

by Dr. Rick Boatright, author of "Surviving Type II Diabetes"
My wife, Linda, is a clinical laboratory scientist who spent 20 years teaching doctors, nurses and scientists about laboratory sciences and managing labs in Arizona and Oregon. When we discovered she was Type 2 Diabetic in 2003, she didn’t care about what “authorities” said about it, she wanted numbers. We researched for more than three years and discovered that the vast majority of the healthcare profession has a very poor understanding of what it is and what causes it.
Type 1 is when the pancreas fails to produce insulin. Type 2, however, is almost completely opposite. Here’s how that happens.
The American diet is actually a process of profound carbohydrate abuse, pure and simple. For those who develop Type 2 Diabetes, the body begins to anticipate the presence of so many carbs and overreacts immediately with very large amounts of insulin to prevent the blood sugar from going too high. It’s actually a survival mechanism. The Type 1 diabetic can’t produce enough insulin, the Type 2 patient produces way too much. Insulin is what takes blood sugar (glucose) out of the blood and takes it into the cells where it’s converted to energy. The energy that’s not used is stored as fat. As the process progresses, the body begins to release too much insulin and the blood sugar goes too low. That’s called Hypoglycemia – not enough blood sugar. This is why hypoglycemia is associated with “pre-diabetes.”
When the blood sugar is low, we’re hungry, so pre-diabetics are always hungry and can often put on a lot of weight (although not always). The extra weight is NOT what CAUSES the diabetes however. What’s causing the diabetes is also what’s causing the excess weight. One does not cause the other.
Soon the body adapts to too much blood sugar entering the cells. Too much sugar can actually burst a cell in a process called “lysis.” This adaptation is known as cell resistance. The cells become resistant to the insulin so the insulin can’t carry in too much blood sugar. This can cause more hunger but at the same time, for some, it can cause the unexplained sudden weight loss associated with Type 2.
When the cells become resistant, the pancreas has to produce even greater amounts of insulin to get blood sugar into the cells and lower the blood sugar. Insulin damages arteries and leaving the person vulnerable to the cardiovascular challenges associated with diabetes.
People who are first diagnosed with Type 2 have had it for an average of seven years prior to diagnosis because it’s so insidious. Blood sugar readings can remain within the normal range for years but only because of the massive amounts of inulin being produced. So casually giving insulin to a Type 2 Diabetic can actually make it WORSE by increasing cell resistance even more.
Eventually, the Type 2 Diabetic’s pancreas can exhaust losing its ability to keep up with insulin demands and the blood sugar skyrockets. These are the advanced patients who, even though they are Type 2 patients who can usually control it with diet, find that they need to begin injecting insulin. However, at this point, technically, the patient goes from being Type 2 to being Type 1, although few doctors understand that point. For a more complete discussion of the subject, refer to my book, “Surviving Type II Diabetes” available at

Misdiagnosing Type 2 Diabetes

Misdiagnosing Type 2 Diabetes:

by Dr. Rick Boatright, author of "Surviving Type II Diabetes"
Type 2 Diabetes has become so prevalent in the United States that it has become one of the major income resources for drug companies and medical supplies companies. In my own estimation, the overabundance of carbohydrates in the American diet is the major cause.
In American, our diet is mainly comprised of foods that grow fast, store for long periods of time and are cheap to produce. In a word – carbohydrates. If the truth be known, the American diet is actually a stark, classic model of carbohydrate ABUSE! It sets up a series of adaptations in the body that are for the purpose of protecting us against that abuse, but become what we know as Type 2 Diabetes. Constant eating of high carb foods causes hyperinsulinemia (excess insulin production) which causes cell resistance which results in chronic high blood sugar and eventual spilling of sugar into the urine.
It is this last symptom that differentiates hyperglycemia from Type 2 Diabetes. When I was in school, the diagnostic criteria for hyperglycemia stated that normal levels of blood glucose (blood sugar) were from 70 to 120 and from 120 to 180 was considered borderline. At 180, the doctor was encouraged to look for diagnostic criteria for possible Type 2 Diabetes. The pathognomic criterion (absolute identifier) was sugar in the urine. If there is no sugar in the urine, it’s NOT actually diabetes, no matter how high the blood sugar reading might read. High blood sugar is hyperglycemia – excess sugar in the blood.
In the diabetic, the kidneys simply cannot handle the hyperglycemia anymore and excess sugar is actually passes into the urine from the blood as it filters though the kidneys.
Even though hyperglycemia may well be a warning sign that a person may be PRE-diabetic, high blood sugar is not the same as diabetes! Hyperglycemia is hyperglycemia. Diabetes is diabetes. They are two separate entities.
What’s my point? Your health record is becoming more and more of a public document. What’s in your record will determine the insurance premiums you must pay and the care that insurers can legally deny. It can affect other areas of your life too if you seek employment or other activities where your health picture can be a consideration.
If your doctor says you have a blood sugar level of some number higher than 120 or 180 and then tells you that you have Type 2 Diabetes, your immediate response must be, “Oh, there’s sugar in my urine?” If there is not, or if the doctor doesn’t know, DEMAND that no diagnosis of diabetes go into your health record until they can positively identify sugar in your urine! Don’t back down. It’s YOUR record. You must make sure it’s accurate.
Hyperglycemia can nearly always be controlled with diet. Often Type2 Diabetes can too. The longer you stay off of drugs, the better off you’ll be. For more details on the subject, read my book, “Surviving Type II Diabetes” available on

When You Crash in Mid-Afternoon

When You Crash in the Mid-Afternoon

by Dr. Rick Boatright, author of "Surviving Type II Diabetes"
Quite often, I have patients who tell me that they have energy enough in the mornings, but by mid-afternoon, they crash!
The primary cause of the mid-afternoon crash is diet-related and is due to having carbohydrates for breakfast and no protein. Let’s look at how that happens:
When we wake in the mornings, we break an 8 to 12-hour fast. That’s why it’s called “breakfast.” So what we put into our empty stomach is important and requires planning based on our goals for the day.
Carbohydrates are very simple nutritional molecules. Because of that, they break down in the stomach (digest) very quickly and subsequently produce energy very quickly. Unfortunately, they also burn up very quickly and are gone. The digestion process takes only about two hours. The insulin triggered by the presence of the carbohydrates carries the blood sugar from the carbohydrates into the cells and your energy is gone by mid-afternoon!
Even though the quick energy may be convenient for getting us up and out of the house, it’s not a good strategy for getting us through the day with even energy reserves.
One way to find a proven successful breakfast strategy for more even energy all day long is to look at the breakfasts of people who absolutely must have energy reserves all day long; people like farmers who work from daylight to dark.
These people typically eat protein for breakfast, things like bacon or sausage and eggs. In Japan, it’s fish. This makes perfect sense because protein is an incredibly long, convoluted and complicated nutritional molecule compared to carbohydrates and correspondingly requires three to four times as long to digest and deliver its energy reserves into the body. The result is long-term, even energy throughout the day with no mid-afternoon crash!
Grains are in fact the worst offender in our American carbohydrate-abuse society. Wheat, in particular, floods blood sugar into the blood stream faster than table sugar and causes a feeling of “logginess” that one may not even realize until they’ve avoided all wheat products and baked goods for a few weeks. Eating a sandwich, for example, with two slices of bread is equivalent to eating a candy bar!
So try some bacon and eggs, some cheese or a little steak or some fish for breakfast and avoid the toast and juice. If you must have juice, make it real grapefruit juice and only an ounce or so. Stay away from the bananas, fruit smoothies, cinnamon toast and pastries. And if you do decide you must indulge in a tasty carb breakfast, just do it on a day when you can take a nap in the afternoon. The predictability is the key.

When a Joint "Pops"

When a Joint “Pops”

by Dr. Rick Boatright, author of "Surviving Type II Diabetes"
I’ve been adjusting joints (including all of the joints in the spine) with an instrument since 1987. When I started chiropractic school, I certainly had no intention of doing that. I wanted to pop and crack people. It felt great! There was an amazing satisfaction to it. But I also became a chiropractor because I wanted to help people as much as I possibly could.
Here’s what I learned about popping joints: There is nitrogen in the synovial fluid that fills the joint capsule in most joints in the body, called synovial joints. When the joint is distracted (“stretched” if you will) as happens when the chiropractor thrusts on the joint, it creates a negative pressure inside the synovial capsule. This negative pressure causes the nitrogen in the synovial fluid to “jump” from a liquid state to a gaseous state; much like the carbon dioxide in a soft drink remains liquid until you remove the bottle cap. But the nitrogen doesn’t bubble, it all jumps at once and creates a “pop.”
So the pop that we hear has nothing whatsoever to do with the joint moving or “popping back into place.” It’s only nitrogen changing its state.
Secondly, in order for that pop of nitrogen to occur, the negative pressure has to be present in the joint capsule for a minimum of 25 milliseconds, 25 one-thousandths of a second. That’s how long it takes to “pop” a joint by hand. It turns out that the fastest reflex mechanism in the body is also about 25 milliseconds.
One of the more important aspects of an effective chiropractic adjustment is speed. The object is to place a thrust into the connective tissue surrounding the joint so fast that the nervous system doesn’t really have a chance to have a reflex reaction to it before it is completed.
Here is where instrument adjusting shines and why the patient doesn’t hear the joint “pop” when the doctor uses an Activator or an Impulse instrument. These instruments deliver their thrusts in about four milliseconds. The entire thrust is initiated, delivered and done about 21 milliseconds before the body realizes that anything has happened and before the nitrogen could possibly leap from liquid to a gas.
In fact, in the 1990’s students in Sweden volunteered to have stainless steel Steinman pins inserted into their spinal bones. Chiropractic thrusts were delivered by hand and with an instrument and the motion was measured with laser accelerometers. Measurements showed that the instruments moved the vertebrae FARTHER in relationship to the ones next to them than did the hand-delivered thrusts. It could be compared to trying to push a nail into a board with the palm of your hand as opposed to pounding it in with a hammer. Even a small hammer is obviously more effective.
So as a chiropractor, I decided to adopt what I believe is the most effective form of spinal correction - instrument adjusting. I’ve never regretted it. If you’re looking for the most excellent chiropractic you can find, be sure to include a good look at instrument adjusting – Activator or Impulse

When Things Go Numb

When Things Go Numb

by Dr. Rick Boatright, author of "Surviving Type II Diabetes"
On my first day of chiropractic school back in 1985, the president of the school addressed the new students. He said something during his address that shocked me at the time, but I later discovered that it is absolutely and unequivocally true!
Your hand or your legs do NOT go numb from a lack of blood flow!!!
I know, you’ve been hearing this erroneous assumption from your parents, your teachers and even from ill-informed doctors throughout your lifetime. The truth is that the one and only kind of tissue in your entire body that conducts sensations is nerve tissue. Not only that, but only half of the nerves in our body conduct any sensations at all, the rest give commands only and feel absolutely nothing.
Let’ look at this rationally. If a lack of blood flow were the real reason for numbness, whatever was numb would quickly become cyanotic. In other words, because there couldn’t be a free exchange of oxygen and carbon dioxide in the local cells, the area would become blue or purple consistent with a build-up of carbon dioxide. It would also feel extremely cold. And because there could be no exchange of nutrients and waste products in the static blood, the numb tissue would begin to die in a matter of minutes.
But this simply doesn’t happen when your arm “goes to sleep,” does it?
People try to justify their assumptions about blood flow by pointing out that there is pressure above the numbness and when the pressure is released, the feeling returns. But that’s like saying that the sun always rises in the East and sets in the West; therefore, the sun orbits the earth every 24 hours. It may be the right observations but it’s certainly the wrong conclusion.
Nerve axons are incredibly long. A substance called “axoplasm” must flow along the nerve axon for it to conduct sensations. If the flow of that axoplasm is restricted, the nerve can’t function. That’s exactly what numbness is – a sensory nerve failing to function.
A phenomenon called “denervation super sensitivity” also proves that the phenomenon is neural and not blood related. When a nerve loses function and then starts to regain function, it not only regains its ability to feel but actually goes overboard creating super sensations. When the feeling comes back into your arm after it goes to sleep, the arm doesn’t simply regain feeling but goes through this period of denervation super sensitivity that we all know as “pins and needles.”
So, get the idea of decreased blood flow causing numbness completely out of your head. It’s right up there with flat earth thinking! Rather, it’s a phenomenon of nerve conduction being impeded. Feel free to correct your doctor if he or she parrots this old wives’ tale too!

How to Build a Triglyceride

How to Build a Triglyceride (and What It Becomes Then)

by Dr. Rick Boatright, author of "Surviving Type II Diabetes"
A patient asked Linda today about how to reduce triglycerides, but like many people, she wasn’t nearly as open to the answer as she thought she would be.
A triglyceride is a long molecular chain comprised of a combination of lipids (fats) and sugars (carbohydrates). In order to build a triglyceride, the body has to have that very combination available – carbohydrates and fats.
With this information, the medical and nutritional gurus have been urging the restriction of fats for decades. But they’ve forgotten why. They only address half of the equation.
It seems inconceivable to them that anyone would voluntarily restrict their carbohydrates. The American diet is a dedication to carbohydrate abuse. And while a low-fat diet may be capable of reducing triglycerides, it’s just as likely that people will still continue to combine what fats they do eat with their carbohydrates, even if it’s unconsciously.
The answer to the above patient’s question was that if you are going to eat carbohydrates, don’t eat any fat with them. If you’re going to eat fats, don’t eat any carbohydrates with them. Of course the automatic objections, indicative of food addictions, immediately pour forth. “I could never eat my eggs without toast! What do I put my hot dog or hamburger on? A steak’s not a steak without a baked potatoes! What about bread and butter,” and the objections continue.
Have a thick, juicy steak and a salad. But don’t eat the potato at the same meal. Have a nice big baked potato with a salad, but not with butter or sour cream. Take the hamburger or hot dog off the bun and feed the bun to the birdies. Never have fries with the hamburger. In fact never have fried fries at all. Have baked “fries,” with a salad, but not with any kind of meat. (Meat always has fat.) A donut is a triglyceride waiting to happen!
Have carbohydrates or have fats, but not both together. The combination is THE requirement for triglycerides! The triglycerides are the reason for the vilification of fats. But nobody is talking about the carbohydrates’ contribution to the building of triglycerides. As a society, we’re too addicted to our national, casual, carbohydrate abuse to touch that sacred cow.
The danger with triglycerides (not fats) is that they become low density lipoproteins, LDLs, the “bad” cholesterol which has been associated, rightly or wrongly, with cardio-vascular diseases. But that’s a discussion for another entire post.

Going Against the Grain!

Going Against the Grain

by Dr. Rick Boatright, author of "Surviving Type II Diabetes"

For some years, Linda and I have felt like a voice in the wilderness. In 2006, I published a book called “Surviving Type II Diabetes” which mentioned many of the health threats presented by grains in general and wheat, specifically. In the February 1, 2012 issue of “Bottom Line Personal,” a 32-year continuous publication claiming to be “The World’s Best Inside Information,” Dr. William Davis gives us the same profound warnings.
Dr. Davis is a preventive cardiologist and director of Track Your Plaque, an interventional heart disease program. He is the author of “Wheat Belly: Lose Wheat, Lose Weight, and Find Your Path Back to Health.” Let me summarize some of his warnings, cautions we have been giving readers and patients for more than a decade:
Like us, he declares that the advice to “Eat more Grains,” is bad advice. He says that that’s not just white bread but the whole grain bread as well. The evidence shows that bread – all of it – increases blood sugar (glucose) MORE than pure sugar! Two slices of bread (a sandwich perhaps) is the equivalent of eating a candy bar. Isn’t that a scary thought?!
He points out that a high-wheat diet is associated with long-term degenerative diseases such as obesity, digestive diseases, arthritis, dementia and heart disease.
Linda and I have come to be able to visually identify high consumers of grains from a distance by their whole-body puffy appearance, especially in the face and around the tummy and “love handles.”
Dr. Davis points out, as we have for so long, that the grains today, especially wheat, have been so hybridized and engineered that they only vaguely resemble grains of yesteryear. The new strains have created proteins that the human body is not designed to handle.
Of particular interest to type two diabetics, it quickly spikes blood sugar, and promotes insulin resistance – the two most powerful threats to the diabetic. It weakens bones and causes increases to LDLs (the “bad” cholesterol).
One of the most immediately favorable aspects of eliminating grains, from the diet, especially wheat, was demonstrated in an experiment at the University of Iowa by the Mayo Clinic. They put 215 obese patients on a wheat free diet and they lost an average of 30 pounds in six months! So even if one people aren’t all that interested in living a long and healthy life, they can certainly get into looking better!
The bottom line is cut the grains, despite all of the “six gains a day” advice. You’ll be amazed at how much better you feel and how much better you’ll be able to control your weight.

Flu Vaccination Considerations

by Dr. Rick Boatright, author of "Surviving TYpe II Diabetes"

The first thing to understand is that there is no such thing as injecting anybody with immunity to disease with the possible exception of immunoglobulin in certain cases of hepatitis.  Even in those cases, however, the injection is not preventative but therapeutic, administered only after the disease has already manifested.

Vaccinations, whether administered by injection, by scratching the skin or by oral dose, are a technique of introducing disease organisms into the body.  Most of the organisms are either dead or “attenuated,” (weakened).  But some are actually live organisms such as one of the polio vaccines.

The introduction of the disease organism into a healthy body causes the person’s immune system to recognize a new invader and to manufacture a new antibody to fight that specific disease organism.  Each disease organism is unique and requires a unique antibody to kill it.  Once the body has dealt with a specific disease organism, the immune system recognizes it from past encounters and produces large amounts of antibodies to attack it before it can become a full-blown disease.

Some vaccinations are effective with a single dose like smallpox.  Others require multiple doses or occasional re-infection (vaccination), such as polio or tetanus.

This brings us to the flu.  The flu is caused by a family of influenza viruses.  These flu viruses are incredibly tiny, and like all things, want to live at any cost.  So their immune systems consist of being able to change their molecular structure just enough so that the body no longer recognizes it and by the time the person’s immune system has built a new immunity to it, the person has gone through two days to two months of illness.

Flu viruses are extremely adept at making their molecular changes quickly, within a matter of months.  This is why you hear about the bird flu one year, the swine flu the next and who knows what the year after that.

When a new flu appears, the vaccination industry must identify it, isolate it, reproduce it, attenuate it and test it for safety before it’s ready to take to the public.  Accomplishing this process in a year or less is an incredibly fast process.  But by then, the flu virus that these new vaccines address have already mutated again, minimizing or completely negating the effectiveness of the vaccine.  In other words, in any present year, they’re always vaccinating for the previous year’s flu!

What’s an alternative?  Four to six thousand milligrams of vitamin C daily, eat lots of garlic. During the flu season, use colloidal silver, goldenseal and Echinacea. Do regular exercise, lower your stress and keep a positive attitude – anything you can do to boost your immune system.  And AVOID SPLENDA as if it were eating the flu itself!!!  It suppresses your immune system by shrinking your thymus gland, especially with long-term use.